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u-shaped microdialysis probe spectra/por rc hollow fibers  (Spectrum Medical Inc)

 
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    Spectrum Medical Inc u-shaped microdialysis probe spectra/por rc hollow fibers
    Ruthenium red inhibited vasopressin neuron activity in non-pregnant, late-pregnant, and lactating rats. ( A – C ) Ratemeter recordings (in 30 s bins) of the firing rates of vasopressin neurons in non-pregnant ( A ), late-pregnant ( B ), and lactating ( C ) rats before and during <t>microdialysis</t> administration of ruthenium red (RuR; 10 mM). ( D ) Mean firing rate (in 10 min bins ± SEM) of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats before and during microdialysis administration of ruthenium red (10 mM). Ruthenium red induced a similar reduction in the firing rate of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats (REPRODUCTIVE STATUS: F 2,28 = 0.69, p = 0.51; TIME: F 6,28 = 5.94, p < 0.001; interaction between REPRODUCTIVE STATUS and TIME: F 12,32 = 0.29, p = 0.99, two-way repeated measures ANOVA). * p < 0.05 and ** p < 0.01 compared to pre-RuR within TIME, Holm–Sidak post hoc tests. ( E – G ) Mean (±SEM) peak early hazard ( E ), late hazard ( F ), and peak early/mean late hazard ratio ( G ) in vasopressin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTUVE STATUS or TIME on the peak early hazard (F 1,28 = 1.70, p = 0.20), mean late hazard (F 1,26 = 0.37, p = 0.85), or hazard ratio (F 1,26 = 0.21, p = 0.81) of vasopressin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME. ( H ). Mean (±SEM) plasma osmolality in non-pregnant, late-pregnant, and lactating rats. Plasma osmolality was affected by reproductive status ( H = 20.0, p < 0.001). * p < 0.05 and ** p < 0.01. Dunn’s post hoc tests compared to non-pregnant rats.
    U Shaped Microdialysis Probe Spectra/Por Rc Hollow Fibers, supplied by Spectrum Medical Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/u-shaped+microdialysis+probe/u+shaped+microdialysis+probe+spectra+por+rc+hollow+fibers/pmc08268815-106-11-24
    Average 90 stars, based on 1 article reviews
    u-shaped microdialysis probe spectra/por rc hollow fibers - by Bioz Stars, 2026-09
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    Images

    1) Product Images from "Plasticity in Intrinsic Excitability of Hypothalamic Magnocellular Neurosecretory Neurons in Late-Pregnant and Lactating Rats"

    Article Title: Plasticity in Intrinsic Excitability of Hypothalamic Magnocellular Neurosecretory Neurons in Late-Pregnant and Lactating Rats

    Journal: International Journal of Molecular Sciences

    doi: 10.3390/ijms22137140

    Ruthenium red inhibited vasopressin neuron activity in non-pregnant, late-pregnant, and lactating rats. ( A – C ) Ratemeter recordings (in 30 s bins) of the firing rates of vasopressin neurons in non-pregnant ( A ), late-pregnant ( B ), and lactating ( C ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( D ) Mean firing rate (in 10 min bins ± SEM) of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats before and during microdialysis administration of ruthenium red (10 mM). Ruthenium red induced a similar reduction in the firing rate of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats (REPRODUCTIVE STATUS: F 2,28 = 0.69, p = 0.51; TIME: F 6,28 = 5.94, p < 0.001; interaction between REPRODUCTIVE STATUS and TIME: F 12,32 = 0.29, p = 0.99, two-way repeated measures ANOVA). * p < 0.05 and ** p < 0.01 compared to pre-RuR within TIME, Holm–Sidak post hoc tests. ( E – G ) Mean (±SEM) peak early hazard ( E ), late hazard ( F ), and peak early/mean late hazard ratio ( G ) in vasopressin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTUVE STATUS or TIME on the peak early hazard (F 1,28 = 1.70, p = 0.20), mean late hazard (F 1,26 = 0.37, p = 0.85), or hazard ratio (F 1,26 = 0.21, p = 0.81) of vasopressin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME. ( H ). Mean (±SEM) plasma osmolality in non-pregnant, late-pregnant, and lactating rats. Plasma osmolality was affected by reproductive status ( H = 20.0, p < 0.001). * p < 0.05 and ** p < 0.01. Dunn’s post hoc tests compared to non-pregnant rats.
    Figure Legend Snippet: Ruthenium red inhibited vasopressin neuron activity in non-pregnant, late-pregnant, and lactating rats. ( A – C ) Ratemeter recordings (in 30 s bins) of the firing rates of vasopressin neurons in non-pregnant ( A ), late-pregnant ( B ), and lactating ( C ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( D ) Mean firing rate (in 10 min bins ± SEM) of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats before and during microdialysis administration of ruthenium red (10 mM). Ruthenium red induced a similar reduction in the firing rate of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats (REPRODUCTIVE STATUS: F 2,28 = 0.69, p = 0.51; TIME: F 6,28 = 5.94, p < 0.001; interaction between REPRODUCTIVE STATUS and TIME: F 12,32 = 0.29, p = 0.99, two-way repeated measures ANOVA). * p < 0.05 and ** p < 0.01 compared to pre-RuR within TIME, Holm–Sidak post hoc tests. ( E – G ) Mean (±SEM) peak early hazard ( E ), late hazard ( F ), and peak early/mean late hazard ratio ( G ) in vasopressin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTUVE STATUS or TIME on the peak early hazard (F 1,28 = 1.70, p = 0.20), mean late hazard (F 1,26 = 0.37, p = 0.85), or hazard ratio (F 1,26 = 0.21, p = 0.81) of vasopressin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME. ( H ). Mean (±SEM) plasma osmolality in non-pregnant, late-pregnant, and lactating rats. Plasma osmolality was affected by reproductive status ( H = 20.0, p < 0.001). * p < 0.05 and ** p < 0.01. Dunn’s post hoc tests compared to non-pregnant rats.

    Techniques Used: Activity Assay, Clinical Proteomics

    Ruthenium red did not affect oxytocin neuron activity in non-pregnant and late-pregnant rats. ( A , B ) Ratemeter recordings (in 30 s bins) of the firing rates of oxytocin neurons in non-pregnant ( A ) and late-pregnant ( B ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( C ) Mean firing rate (in 10 min bins ± SEM) of oxytocin neurons in non-pregnant and late-pregnant rats before and during microdialysis administration of ruthenium red (10 mM). Oxytocin neuron activity was not affected by ruthenium red in non-pregnant or late-pregnant rats (REPRODUCTIVE STATUS: F 1,9 = 0.03, p = 0.87; TIME: F 6,9 = 1.10, p = 0.37; interaction between REPRODUCTIVE STATUS and TIME: F 6,9 = 1.00, p = 0.41, two-way repeated measures ANOVA). ( D – F ) Mean (±SEM) peak early hazard ( D ), late hazard ( E ), and peak early/mean late hazard ratio ( F ) in oxytocin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTIVE STATUS or TIME on the peak early hazard (F 1,9 = 0.58, p = 0.46), mean late hazard (F 1,9 = 1.97, p = 0.19), or hazard ratio (F 1,9 = 0.09, p = 0.77) of oxytocin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME.
    Figure Legend Snippet: Ruthenium red did not affect oxytocin neuron activity in non-pregnant and late-pregnant rats. ( A , B ) Ratemeter recordings (in 30 s bins) of the firing rates of oxytocin neurons in non-pregnant ( A ) and late-pregnant ( B ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( C ) Mean firing rate (in 10 min bins ± SEM) of oxytocin neurons in non-pregnant and late-pregnant rats before and during microdialysis administration of ruthenium red (10 mM). Oxytocin neuron activity was not affected by ruthenium red in non-pregnant or late-pregnant rats (REPRODUCTIVE STATUS: F 1,9 = 0.03, p = 0.87; TIME: F 6,9 = 1.10, p = 0.37; interaction between REPRODUCTIVE STATUS and TIME: F 6,9 = 1.00, p = 0.41, two-way repeated measures ANOVA). ( D – F ) Mean (±SEM) peak early hazard ( D ), late hazard ( E ), and peak early/mean late hazard ratio ( F ) in oxytocin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTIVE STATUS or TIME on the peak early hazard (F 1,9 = 0.58, p = 0.46), mean late hazard (F 1,9 = 1.97, p = 0.19), or hazard ratio (F 1,9 = 0.09, p = 0.77) of oxytocin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME.

    Techniques Used: Activity Assay

    Related Articles

    other:

    Article Title: Attenuated hypothalamic responses to α‐melanocyte stimulating hormone during pregnancy in the rat
    Article Snippet: Following removal of the meninges, a U‐shaped microdialysis probe (in‐house design, supplied by Prof. M. Ludwig, University of Edinburgh; total membrane length 2.0 mm, permeable to 10 kDa; Spectra/Por RC Hollow Fibres, Spectrum Medical Inc., Houston, TX, USA) was bent (between 100 and 200 deg) and the loop of the membrane was positioned to lie flat over the exposed ventral surface of the brain over the SON.

    Article Title: A switch from GABA inhibition to excitation of vasopressin neurons exacerbates the development angiotensin II-dependent hypertension.
    Article Snippet: Hypothalamic magnocellular neurons secrete vasopressin into the systemic circulation to maintain blood pressure by increasing renal water reabsorption and by vasoconstriction.. When blood pressure rises, baroreflex activation normally inhibits vasopressin neurons via activation of GABAergic inputs.. However, plasma vasopressin levels are paradoxically elevated in several models of hypertension and in some patients with essential hypertension, despite increased blood pressure.

    Article Title: Glial regulation of extrasynaptic NMDA receptor-mediated excitation of supraoptic nucleus neurones during dehydration.
    Article Snippet: Correspondence to: Colin H. Brown, Department of Physiology, University of Otago, PO Box 56, Dunedin 9054, New Zealand (e-mail: colin.brown@otago.ac.nz).. Magnocellular neurosecretory cells (MNCs) of the supraoptic nucleus (SON) project to the posterior pituitary gland where they release the hormones, vasopressin and oxytocin into the circulation to maintain plasma osmolality.. Hormone release is proportionate to SON MNC action potential (spike) firing rate.



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    90
    Eicom Corporation u-shaped microdialysis probe
    Ruthenium red inhibited vasopressin neuron activity in non-pregnant, late-pregnant, and lactating rats. ( A – C ) Ratemeter recordings (in 30 s bins) of the firing rates of vasopressin neurons in non-pregnant ( A ), late-pregnant ( B ), and lactating ( C ) rats before and during <t>microdialysis</t> administration of ruthenium red (RuR; 10 mM). ( D ) Mean firing rate (in 10 min bins ± SEM) of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats before and during microdialysis administration of ruthenium red (10 mM). Ruthenium red induced a similar reduction in the firing rate of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats (REPRODUCTIVE STATUS: F 2,28 = 0.69, p = 0.51; TIME: F 6,28 = 5.94, p < 0.001; interaction between REPRODUCTIVE STATUS and TIME: F 12,32 = 0.29, p = 0.99, two-way repeated measures ANOVA). * p < 0.05 and ** p < 0.01 compared to pre-RuR within TIME, Holm–Sidak post hoc tests. ( E – G ) Mean (±SEM) peak early hazard ( E ), late hazard ( F ), and peak early/mean late hazard ratio ( G ) in vasopressin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTUVE STATUS or TIME on the peak early hazard (F 1,28 = 1.70, p = 0.20), mean late hazard (F 1,26 = 0.37, p = 0.85), or hazard ratio (F 1,26 = 0.21, p = 0.81) of vasopressin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME. ( H ). Mean (±SEM) plasma osmolality in non-pregnant, late-pregnant, and lactating rats. Plasma osmolality was affected by reproductive status ( H = 20.0, p < 0.001). * p < 0.05 and ** p < 0.01. Dunn’s post hoc tests compared to non-pregnant rats.
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    Ruthenium red inhibited vasopressin neuron activity in non-pregnant, late-pregnant, and lactating rats. ( A – C ) Ratemeter recordings (in 30 s bins) of the firing rates of vasopressin neurons in non-pregnant ( A ), late-pregnant ( B ), and lactating ( C ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( D ) Mean firing rate (in 10 min bins ± SEM) of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats before and during microdialysis administration of ruthenium red (10 mM). Ruthenium red induced a similar reduction in the firing rate of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats (REPRODUCTIVE STATUS: F 2,28 = 0.69, p = 0.51; TIME: F 6,28 = 5.94, p < 0.001; interaction between REPRODUCTIVE STATUS and TIME: F 12,32 = 0.29, p = 0.99, two-way repeated measures ANOVA). * p < 0.05 and ** p < 0.01 compared to pre-RuR within TIME, Holm–Sidak post hoc tests. ( E – G ) Mean (±SEM) peak early hazard ( E ), late hazard ( F ), and peak early/mean late hazard ratio ( G ) in vasopressin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTUVE STATUS or TIME on the peak early hazard (F 1,28 = 1.70, p = 0.20), mean late hazard (F 1,26 = 0.37, p = 0.85), or hazard ratio (F 1,26 = 0.21, p = 0.81) of vasopressin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME. ( H ). Mean (±SEM) plasma osmolality in non-pregnant, late-pregnant, and lactating rats. Plasma osmolality was affected by reproductive status ( H = 20.0, p < 0.001). * p < 0.05 and ** p < 0.01. Dunn’s post hoc tests compared to non-pregnant rats.

    Journal: International Journal of Molecular Sciences

    Article Title: Plasticity in Intrinsic Excitability of Hypothalamic Magnocellular Neurosecretory Neurons in Late-Pregnant and Lactating Rats

    doi: 10.3390/ijms22137140

    Figure Lengend Snippet: Ruthenium red inhibited vasopressin neuron activity in non-pregnant, late-pregnant, and lactating rats. ( A – C ) Ratemeter recordings (in 30 s bins) of the firing rates of vasopressin neurons in non-pregnant ( A ), late-pregnant ( B ), and lactating ( C ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( D ) Mean firing rate (in 10 min bins ± SEM) of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats before and during microdialysis administration of ruthenium red (10 mM). Ruthenium red induced a similar reduction in the firing rate of vasopressin neurons in non-pregnant, late-pregnant, and lactating rats (REPRODUCTIVE STATUS: F 2,28 = 0.69, p = 0.51; TIME: F 6,28 = 5.94, p < 0.001; interaction between REPRODUCTIVE STATUS and TIME: F 12,32 = 0.29, p = 0.99, two-way repeated measures ANOVA). * p < 0.05 and ** p < 0.01 compared to pre-RuR within TIME, Holm–Sidak post hoc tests. ( E – G ) Mean (±SEM) peak early hazard ( E ), late hazard ( F ), and peak early/mean late hazard ratio ( G ) in vasopressin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTUVE STATUS or TIME on the peak early hazard (F 1,28 = 1.70, p = 0.20), mean late hazard (F 1,26 = 0.37, p = 0.85), or hazard ratio (F 1,26 = 0.21, p = 0.81) of vasopressin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME. ( H ). Mean (±SEM) plasma osmolality in non-pregnant, late-pregnant, and lactating rats. Plasma osmolality was affected by reproductive status ( H = 20.0, p < 0.001). * p < 0.05 and ** p < 0.01. Dunn’s post hoc tests compared to non-pregnant rats.

    Article Snippet: Following the removal of the meninges overlying the supraoptic nucleus, a U-shaped microdialysis probe [ ], permeable to 10 kDa (Spectra/Por RC Hollow Fibers, Spectrum Medical Inc., Houston, TX, USA), was bent to position the loop of the membrane over the ventral surface of the supraoptic nucleus.

    Techniques: Activity Assay, Clinical Proteomics

    Ruthenium red did not affect oxytocin neuron activity in non-pregnant and late-pregnant rats. ( A , B ) Ratemeter recordings (in 30 s bins) of the firing rates of oxytocin neurons in non-pregnant ( A ) and late-pregnant ( B ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( C ) Mean firing rate (in 10 min bins ± SEM) of oxytocin neurons in non-pregnant and late-pregnant rats before and during microdialysis administration of ruthenium red (10 mM). Oxytocin neuron activity was not affected by ruthenium red in non-pregnant or late-pregnant rats (REPRODUCTIVE STATUS: F 1,9 = 0.03, p = 0.87; TIME: F 6,9 = 1.10, p = 0.37; interaction between REPRODUCTIVE STATUS and TIME: F 6,9 = 1.00, p = 0.41, two-way repeated measures ANOVA). ( D – F ) Mean (±SEM) peak early hazard ( D ), late hazard ( E ), and peak early/mean late hazard ratio ( F ) in oxytocin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTIVE STATUS or TIME on the peak early hazard (F 1,9 = 0.58, p = 0.46), mean late hazard (F 1,9 = 1.97, p = 0.19), or hazard ratio (F 1,9 = 0.09, p = 0.77) of oxytocin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME.

    Journal: International Journal of Molecular Sciences

    Article Title: Plasticity in Intrinsic Excitability of Hypothalamic Magnocellular Neurosecretory Neurons in Late-Pregnant and Lactating Rats

    doi: 10.3390/ijms22137140

    Figure Lengend Snippet: Ruthenium red did not affect oxytocin neuron activity in non-pregnant and late-pregnant rats. ( A , B ) Ratemeter recordings (in 30 s bins) of the firing rates of oxytocin neurons in non-pregnant ( A ) and late-pregnant ( B ) rats before and during microdialysis administration of ruthenium red (RuR; 10 mM). ( C ) Mean firing rate (in 10 min bins ± SEM) of oxytocin neurons in non-pregnant and late-pregnant rats before and during microdialysis administration of ruthenium red (10 mM). Oxytocin neuron activity was not affected by ruthenium red in non-pregnant or late-pregnant rats (REPRODUCTIVE STATUS: F 1,9 = 0.03, p = 0.87; TIME: F 6,9 = 1.10, p = 0.37; interaction between REPRODUCTIVE STATUS and TIME: F 6,9 = 1.00, p = 0.41, two-way repeated measures ANOVA). ( D – F ) Mean (±SEM) peak early hazard ( D ), late hazard ( E ), and peak early/mean late hazard ratio ( F ) in oxytocin neurons from non-pregnant, late-pregnant, and lactating rats. There was no effect of REPRODUCTIVE STATUS or TIME on the peak early hazard (F 1,9 = 0.58, p = 0.46), mean late hazard (F 1,9 = 1.97, p = 0.19), or hazard ratio (F 1,9 = 0.09, p = 0.77) of oxytocin neurons during ruthenium red administration, and no interaction between REPRODUCTUVE STATUS and TIME.

    Article Snippet: Following the removal of the meninges overlying the supraoptic nucleus, a U-shaped microdialysis probe [ ], permeable to 10 kDa (Spectra/Por RC Hollow Fibers, Spectrum Medical Inc., Houston, TX, USA), was bent to position the loop of the membrane over the ventral surface of the supraoptic nucleus.

    Techniques: Activity Assay